By Manuel Patarroyo, Lennart Lindbom (auth.), Patrick Y.-K. Wong, Charles N. Serhan (eds.)
This quantity constitutes. partly. the lawsuits of the symposium on "Cell-Cell interplay and liberate of Inflammatory Mediators" prepared through Drs. Patrick Y-K Wong and Charles N. Serhan and awarded on the FASEB assembly in Washington. D.C. in April. 1990. It comprises chapters by way of the symposium audio system in addition to contributions from investigators during this box. Readers will locate interesting advances during this quantity. which includes chapters devoted to state of the art wisdom within the box of Cell-Cell interplay and the capabilities of published mediators in inflammatory ailments. This booklet contains "cutting facet" investigations on transcellular eicosanoid biosynthesis. cytokines. PAF, and adhesion in addition to interactions of inflammatory cells with endothe 1 i um. 1 ung • and kidney. A 1 so. the contro 1 and regu 1 at ion of renal functionality by means of lipid mediators generated in the course of cell-cell i nteracti ons among rena 1 mesangi a 1 cells and 1 eukocytes has generated perception into the mobile biology and regulatory position of those mediators within the kidney. in addition. the connection among those components is mentioned in sequelae of either asthmatic and renal illnesses. we are hoping that the various enthusiasm and pleasure found in this examine also are obvious right here and that this quantity will function a reference for researchers. lecturers. and scholars to survey thi s swiftly transforming into field.
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Extra info for Cell-Cell Interactions in the Release of Inflammatory Mediators: Eicosanoids, Cytokines, and Adhesion
The effect of GM-CSF is rapid and not mediated by protein synthesis nor by protein kinase C (52). It has also been observed that the continuous infusion of GM-CSF into patients rapidly upregulates the expression of granulocyte CD11b (129). A logical consequence of the aforementioned GM-CSF-induced increase in surface expression of certain proteins is the increase of neutrophil adhesiveness. CD11b (Mol), selectively expressed after neutrophils are exposed to GM-CSF, is a member of a family of surface glycoproteins that are essential for adhesion-dependent granulocyte functions.
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