Acute aortic disease by John Elefteriades

By John Elefteriades

Overlaying the pathophysiology, imaging, analysis, and therapy of various aortic aneurysms and dissections, this resource is helping physicians successfully study and overview affected contributors in medical or emergency care settings. delivering a wide range of illustrations, x-rays, and operative photos to stress key anatomic observations, this consultant includes state-of-the-art perception at the most recent biologic, radiologic, scientific, and surgical advancements that experience taken position within the box. offered in a reader-friendly layout, this resource presents end-of-chapter questions and a point-counterpoint structure to investigate differing views from popular specialists on those ailments. The Q

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Grannis FW Jr, Bryant C, Caffaratti JD, et al. Acute aortic dissection associated with cocaine abuse. Clin Cardiol 1988; 11:572–574. 39. Rashid J, Eisenberg MJ, Topol EJ. Cocaine-induced aortic dissection. Am Heart J 1996; 132:1301–1304. 40. Nienaber CA, Eagle KA. Aortic dissection: new frontiers in diagnosis and management. Circulation 2003; 108:628–635 (Pt I), 772–778 (Pt II). 41. Januzzi JL, Isselbacher EM, Fattori R, et al. Characterizing the young patient with aortic dissection: results from the International Registry of Aortic Dissection (IRAD).

33. Januzzi J, Sabatine MS, Eagle KA, et al. Iatrogenic aortic dissection. Am J Cardiol 2002; 89:623–626. indd21 21 2/14/2007 5:51:21 PM 22 Nienaber and Ince 34. Pieters FAA, Widdershoven JW, Gerardy AC, et al. Risk of aortic dissection after aortic valve replacement. Am J Cardiol 1993; 72:1043–1047. 35. Epperlein S, Mohr-Kahaly S, Erbel R, et al. Aorta and aortic valve morphologies predisposing to aortic dissection. An in vivo assessment with transesophageal echocardiography. Eur Heart J 1994; 15:1520–1527.

Thus, PPAR-γ expression might reflect the pathogenesis of cystic medial degeneration and disease progression in the aorta of Marfan’s and nonMarfan’s patients (15). EDS is a heterogeneous group of hereditable connective tissue disorders characterized by articular hypermobility, skin hyperextensibility, and tissue fragility. Eleven types of EDS have been characterized. The true prevalence of EDS is unknown. An aggregate incidence of 1/5000 births is often cited, with no racial or ethnic predisposition.

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